Blow Out the Candles…

If there is any treatment that I feel like gets used in a cookie-cutter fashion, it’s pursed-lip breathing. As much as I hate seeing this technique used for every single patient that has shortness of breath, it does have clinical usefulness. So let’s talk about how to implement pursed-lip breathing properly based on patient presentation and medical history so that you can use this most effectively and appropriately. This is going to take up down some roads, both general and specific, to help you understand what pursed-lip breathing does and how it should be used. But, first, let’s watch a pretty decent video on pursed-lip breathing. This video incorporates several different techniques along with pursed-lip breathing, but doesn’t give a whole lot of clinical specifics which makes it GREAT for patient education. You’ll need to clarify the specifics for your patient based on their needs.

American Lung Association

Let’s start at the beginning: What is Pursed Lip Breathing?
Pursed-lip breathing is a respiratory technique with many uses depending on how it is implemented. The general purpose of pursed lip breathing is to prolong exhalation, slow respiration, and provide positive pressure to breathing. Overall, this technique can improve vital capacity and tidal volume.

Sounds simple enough right? We hear the same cues used all the time: “Smell the roses, blow out the candles,” but I have to tell you… If you think you know, you have no idea. This is so much more complex than we give it credit for. Let’s break this down by diagnosis…


Restrictive lung diseases:
Lung resection, sarcoidosis, chronic atelectasis, pulmonary fibrosis including interstitial lung diseases, COVID-19 and other viral/bacterial pneumonias, pleural effusion

There is no amount of deep, diaphragmatic, or pursed lip breathing you can do that will change the fact that these patients have restricted lung volume. You can’t get air in if there is nowhere to put it, you cannot use what just isn’t there. Pursed lip breathing can still be used for these patients, but the use is NOT to improve vital capacity, because that won’t happen. When people with restrictive lung conditions become short of breath, it is typically associated with a high level of stress and anxiety because they literally can’t breathe. Their lungs are closing down or scarring up or any other mechanism that reduces viable lung tissue amount or function.

Pursed-lip breathing can help these folks, for sure, but it is through mechanisms that are emotionally and autonomically linked to breathing. Mindfulness of breathing has been shown by numerous studies (thank you, yoga and tai chi!) to change a person’s emotional state and physiological function. This mechanism is well studied but very complex and involves chemical transmitters as well as stretch-response mechanisms in the musculoskeletal, vascular, cardiac, olfactory, and limbic systems (and probably several others). It is important to note that the slowed rate of breathing is the emphasis of the research, not necessarily the “pursed-lip” portion of the technique.

Photo by Anna Shvets on Pexels.com

That all being said, please use pursed lip breathing in this individuals for the purpose of decreasing anxiety and respiratory rate. Keep in mind that this may only be a band-aid and that medications, increased oxygen titration, and other medical interventions may be necessary to recover shortness of breath. Pursed lip and diaphragmatic breathing can be used together to stimulate the autonomic nervous system (chemo-, baro-, and stretch-receptors) to reduce respiratory rate and chemically alter emotional state to reduce stress and anxiety and lessen symptoms of shortness of breath. This is based in parasympathetic activation which slows heart rate and relaxes smooth muscles throughout the body. This means arteries relax and are able to carry more oxygen rich blood to needed organ systems, blood pressure lowers, and cardiac effort is reduced due to reduced peripheral resistance. All good things for someone who is experiencing shortness of breath 🙂

Unfortunately, research shows us that pursed lip breathing may also increase the overall metabolic workload of breathing in patients with restrictive lung diseases, which may outweigh the benefits if you are attempting to increase oxygen saturation with activity. It has also been found that pursed lip breathing is not effective to reduce shortness of breath during activity in patients with restrictive lung diseases. So, although it may seem pretty simple, this intervention does need to be used with caution and for the right reasons.


Obstructive Lund Diseases:
Chronic Obstructive Pulmonary Disease, Bronchitis, Bronchiolitis, Emphysema, Cystic Fibrosis, Asthma

This one is the big red flag for me. The video does a pretty decent job of showing how to accommodate pursed-lip breathing to obstructive conditions, but doesn’t exactly explain the reasoning. The big problem with obstructive lung disease is that you can’t get enough air OUT of the lungs. Therefore, telling someone to breathe in deeply through their nose is NOT going to improve their symptoms! In fact, it will actually make things worse because you are just attempting to pack more air in to a space that is already full of dead air. This does not improve ventilation, and significantly reduces ventilation/perfusion matching requirements of activity. In essence, the more you have them breathe deeply, the less oxygen they have left in their blood supply to actually perform the mechanical work of breathing!

To perform pursed lip breathing properly in a patient who has obstructive lung disease, you have to have them focus on the breathing OUT portion of pursed lip breathing. And this activity is two-fold in benefits! Here’s why:

The video instructs pretty well on the importance of breathing out and incorporates a great tool I teach to my patients all the time: Counting! Patients with obstructive lung conditions need to get more air out than they take in. This helps remove the dead air from their lungs and replaces it with new air. The more air they breathe out, the more room they have to put new air in. Make sense? So having your patient count, “1, 2” for their breath IN and “1, 2, 3, 4” for their breath OUT helps them focus on getting the air out. Sometimes I add in a PEP device to help shift to focus to getting air OUT for patients with obstructive conditions.

Photo by Anna Shvets on Pexels.com

The second bonus to focusing on exhalation is the actual performance of the “pursed-lip” portion of pursed-lip breathing. This maneuver, when done properly, causes some of the air coming out of the lungs to bounce off the inside of the lips and cheeks and rebound into the airway and lungs. This is similar to PEEP (positive end-expiratory pressure). We provide PEEP when patients are mechanically ventilated with the exact same purpose it fulfills with pursed lip breathing: to provide back pressure that splints open smaller or collapsed airways, allowing for exhalation of greater volumes of carbon dioxide, therefore improving vital capacity. That means that the little bit of back pressure holds the airways open longer to allow for more dead air to escape, making room for more good air to get back in.

And when I say “done properly,” I don’t mean that you try to force air out through whistle-tight lips. That just doesn’t allow for that back pressure to build up correctly. Just like in mechanical ventilation, too much PEEP can be a problem. Lips should still be open somewhat and the cheeks should be loose and allowed to flare so that air can gather and build pressure. Here’s a terrible up-close video of me pursed-lip breathing so you can get the idea.

Like I said, pretty terrible up-close video, but I’m not pinching my lips together super tight and my cheeks are relaxed and that’s really the point here.

There have been some recent studies that have shown that a simulated pursed-lip breathing during non-invasive ventilation (NIV) can actually be more effective than NIV alone for patients with COPD who are in hypercapnic respiratory failure! This intervention even held up under activity demands of a functional maximal exercise capacity test (the 6 minute walk test).


Did you know there was that much to it? I know I was shocked to find out something that seemed so simple was actually so complex and that it could be used in such varying ways if it was used correctly. I hope that you are able to repurpose this old stand-by technique with better clinical application for your patients.

I’ve gotta tell you all something… I love this stuff. I love getting down to the absolute basics, figuring out the “why” of it all, applying it in the best possible way for my patients, and then telling all of you about it! Now, pucker those lips and get to breathing!


Do you find that pursed-lip breathing recovers shortness of breath better for your restrictive or obstructive patients? Let me know in the comments!

Read more from the Pulmonary Rehab Toolbox!

Postural Drainage

We’ve all seen that dreaded picture in our textbooks… All the human figures laying in so many different positions with pillows and tables tilted all over… and I very clearly remember thinking, “How on earth am I supposed to remember all of those?” Well, good news. You really don’t have to. It’s great if youContinue reading “Postural Drainage”

Spilling the Box of Pearls: All the Tips on Supplemental Oxygen Management

In my recent post on COPD management, I mentioned that there are some really important parts of supplemental oxygen management that you need to be aware of and consider in your practice. If you are assisting patients who utilize supplemental oxygen regularly, you need to keep these things in mind. You also may be workingContinue reading “Spilling the Box of Pearls: All the Tips on Supplemental Oxygen Management”

Chronic Disease Part 2: Chronic Obstructive Pulmonary Disease (COPD)

This is part 2 in a multi-part series on the role of Rehab Providers in the management of chronic disease. Don’t forget to check out Part 1: Heart Failure! Chronic Obstructive Pulmonary Disease is a widely diagnosed disease of the lungs that includes the diagnoses of emphysema and chronic bronchitis. COPD can be caused byContinue reading “Chronic Disease Part 2: Chronic Obstructive Pulmonary Disease (COPD)”

Bellissimo, G., Leslie, E., Maestas, V., & Zuhl, M. (2020). The Effects of Fast and Slow Yoga Breathing on Cerebral and Central Hemodynamics. International journal of yoga13(3), 207–212. https://doi.org/10.4103/ijoy.IJOY_98_19

Jünger, C., Gaede, K. I., Herzmann, C., Lange, C., Reimann, M., Rüller, S. (2020). Pursed-lip breathing ventilation for the treatment of hypercapnic respiratory failure in COPD. ERJ Open Research. 6: 41; DOI: 10.1183/23120541.RFMVC-2020.41

Jünger, C., Rüller, S., Reimann, M., Krabbe, L., Gaede, K., Lange, C., Herzman, C. (2019). Mechanical non-invasive pursed-lip breathing ventilation for hypercapnic patients with COPD. European Respiratory Journal. 54: PA4237; DOI: 10.1183/13993003.congress-2019.PA4237

Nguyen J, Duong H. Pursed-lip Breathing. [Updated 2020 Aug 11]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2020 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK545289/

Parisien-La Salle, S., Rivest-Abel, E., Boucher, V. G., Lalande-Gauthier, M., Poirier, C., Dube, B. P., Manganas, H., Morisset, J., Comtois, A. S. (2017). Effect of pursed lip breathing on dyspnea and walking distance in interstitial lung disease: a randomized crossover study. European Respiratory Journal 50: PA2246; DOI: 10.1183/1393003.congress-2017.PA2246

Russo, M. A., Santarelli, D. M., & O’Rourke, D. (2017). The physiological effects of slow breathing in the healthy human. Breathe (Sheffield, England)13(4), 298–309. https://doi.org/10.1183/20734735.009817

Zaccaro, A., Piarulli, A., Laurino, M., Garbella, E., Menicucci, D., Neri, B., & Gemignani, A. (2018). How Breath-Control Can Change Your Life: A Systematic Review on Psycho-Physiological Correlates of Slow Breathing. Frontiers in human neuroscience12, 353. https://doi.org/10.3389/fnhum.2018.00353

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Emergency Response Screening

I hope, at this point, you’ve all heard of the FAST acronym for identifying signs of a cerebrovascular accident (aka stroke). My in-laws even have a magnet on their fridge with a great comic strip describing the FAST acronym. I have run in to a couple different versions of it in the last few years, but I’ll give you the basic one so you know what I’m talking about. But, we will also go over some other tools you can have in your arsenal for emergency response screening and I’ll tell you where you can go find even more!


I’m not sure if calling 911 is a skill or not, but I’ve certainly done it enough times that I have the process pretty much figured out. We skip the pleasantries and get straight to the point: Where are you, what’s the situation, who is the caller. DON’T WASTE TIME saying “Hi.” Wait your turn to talk. Use speakerphone so you can follow whatever instructions they give you. And always prioritize symptoms: breathing is always MOST important! You are given a priority number (0 is highest priority – that’s for the not breathing folks). And you NEVER hang up until you are told to do so or EMS arrives. It’s fast. It’s nerve-racking the first few times, but you get used to it (that’s a bit weird to say). If you are in doubt, you have two options: Call the physician and request next steps OR

Emergency response is a basic skill for rehabilitation clinicians and Physical Therapists are skilled to provide first line triage in the case of an emergency. Our specialty is movement: to move someone or not to move someone, and how that movement should be done is a critical part of not only our interventions but also emergency response. Large scale emergencies or single patient emergencies both require this level of clinical decision-making. Thankfully, if you don’t feel like you’re ready to provide that knowledge or skill, there are lots of tools to help you in both small and large scale emergencies.

My best advice: Stay calm, trust your training, and trust your gut. If something feels wrong, it probably is.


Let’s look at the FAST tool we mentioned. This is what really got me started down this rabbit hole of exploring emergency response screening processes. What is fast, easy, and gives me the most information to make the determination of what my next step is? The FAST tool is easy and, well… fast. I don’t have to do much other than look at someone and I know they need emergency help. I also know I need to get that help as fast as I can.

American Stroke Association

Pretty self-explanatory. I’ve had a few significant events in my practice (and outside of my practice!) where I have had to utilize this tool and other tools to assess my patients. I’ll tell you a little about these events so you know when and how to use these tools. I hope you can’t implement them quickly and easily so that, no matter what setting you are in, you can determine the best course of action for your patients, family, friends, or random strangers at your friend’s wedding…


The Stroke Diagnostic Tool

One of my patients in an AFC had a history of middle cerebral artery stroke which is what landed him in an AFC to begin with. One day, I showed up for his visit and he had fallen in the bathroom. The door was locked so couldn’t let me in to help him. His staff was unaware this had happened. As I was talking to him through the door, he told me he was uninjured but couldn’t seem to get up. He was confused and his speech was slurring harder than normal. I knew this because I had established a neurological baseline at my first visit with him.

After the AFC staff assisted me in opening the bathroom door, his symptoms persisted, so I took a set of vitals while calling 911. Although his heart rate was a bit high (he did just fall in the bathroom, after all…), his blood pressure was normal and he did have a slight fever. Upon answering and receiving the required information, the Emergency Dispatcher had me preform a stroke screening. In case you’ve never done this or heard of it, it’s three simple questions:

  1. Can he smile? (this checks for facial droop)
  2. Can he raise both arms equally overhead? (this checks for unilateral paresis/paralysis)
  3. Can he say, “The early bird gets the worm?” (this checks for slurring or other speech deficits)

This is called the Medical Priority Dispatch System (MPDS) Stroke Diagnostic Tool (SDxT), and it is based on research of the FAST acronym for stroke screening. The SDxT has a high specificity (0.96) and a not so impressive sensitivity (0.41). Therefore, this tool is great for ruling in a NEW stroke, but, because my patient already had a stroke with residual deficits, it gave false results. I had to repeatedly interpret the results in comparison to his baseline. I’m sure the dispatcher was really annoyed with me saying, “No more than his baseline,” as an answer to her first two questions. Thankfully, it was only a new UTI and he turned out fine after some antibiotics and inpatient rehab. Because this tool has a specificity of 0.96, though, it is great for ruling in new stroke if your patient presents with these clinical symptoms in any setting.

PT Clinical Decision-Making:
Is this an emergency? Yes, single-patient – needs evaluation s/p fall and for neurological changes, FAST criteria met, SDxT inconclusive
Does this patient need to move? Yes
How should this patient move? Lift assist team from the floor, track-chair up the stairs and out of the house (a chair on tracks that can climb stairs), and by ambulance to hospital


Other Clinical Tools

Another piece of the puzzle needs to be blood pressure. However, like I’ve mentioned many times before, taking a single blood pressure measurement (although better than doing nothing) doesn’t give you much information. You need to have a baseline measurement. The rate of noncompliance with hypertension medication can be anywhere from 40-60% depending on the article you read. So, even if you KNOW your patient has hypertension, that doesn’t mean they’ve been managing it properly. Those medications can have some side effects that people just don’t like (like going to the bathroom too often or symptoms of orthostasis), so people tend to not take them for different reasons. And that’s just the people you KNOW about! We’ve talked about exercise induced hypertension which may be present in just about anyone, and we don’t even really know how best to manage that yet. And then there is the basic fact that you can’t just look at someone and know they have hypertension. Clinical symptoms don’t always present in people who are young and healthy otherwise, and they often are missed. Please take a read in the article about blood pressure basics and dig in to some of the items regarding blood pressure management. We, as Physical Therapists, should be playing a crucial role in this arena. This is my soapbox and I will die on it. Vitals are vital.

Photo by cottonbro on Pexels.com

Blood pressure screening isn’t just useful to identify cerebrovascular accidents! If you take that baseline blood pressure and you know they typically run high, maybe 160/94, you may not be as concerned when you see a 166/94 pop up at your visit today. However, if you have established a baseline 160/94 blood pressure and you see 200/106 show up, now you can make a clinical decision regarding what you need to do next: do some further assessment because you know they run high, call the physician to determine next steps if they are exhibiting no symptoms, or call 911 if they are are in a hypertensive crisis with symptoms.

Unfortunately, if you see 164/90 show up, but you have no baseline to compare to, you may not know what to do. One of my very first patients as a PT student presented with a 164/100 blood pressure one day, but was having many symptoms of something else going on. She did not meet FAST criteria and I had no comparison blood pressure to go off of. I had clinical presentation of something so I called 911. She had a seizure in the Emergency Department shortly after arriving, but turns out she had a history of seizures. Hypertension was kind of like an aura for her. You just never know. If I had taken a baseline blood pressure, I may have had a better idea of what to do with her and not struggled to figure it out until the end of our visit.

PT Clinical Decision-Making
Is this an emergency? I didn’t know because I had no baseline to determine if this was a hypertensive crisis for her and I didn’t know her history of seizures. But, No, it was not an emergency due to her history.
Does patient need to move? Yes, but…
How does this patient need to move? …only to the floor if I would have known her seizure history. I would have called the physician for persistent symptoms and notified them of a seizure if it happened. She could have transferred to the hospital via private car with a driver if necessary depending on the outcome and length of the seizure.

To be very clear: DO NOT DELAY CALLING 911 to take a patient’s blood pressure. If they’ve met the FAST criteria, SDxT criteria, both, or ANY other emergency response criteria, and you haven’t taken their blood pressure yet, call for emergency medical assistance first! Time is literally brain cells. Don’t waste even a second. Like my patient above, even if it isn’t a stroke, they may still need medical attention for some reason. You can take their blood pressure while you wait for EMS to arrive or when the Dispatcher asks you to do it.


And then there is my friend’s wedding…

There I am in a gown at the head table during the reception and down goes someone in the crowd. The medical expertise at the reception included me, an NP student, and an ortho nurse. Not your best emergency response team, but better than nothing, right? I rush over to this mildly responsive man who cannot move and is slurring something and I’m immediately informed by a family member that he has a history of stroke somewhat recently. I observed pretty quickly that he met the FAST criteria so, I call for someone to call 911 because time is brain cells here.

Having no equipment other than my brain, I immediately started the SDxT assessment to save time, but again, because he has a history of CVA, lots of positives show up: heavily slurred speech, inability to raise the left arm, and obvious facial droop (I didn’t have to ask him to smile…). I started a detailed neuro assessment (think acute care-style) with his family member assisting for comparison to baseline and sure enough… exacerbation of symptoms. EMS arrived, I handed off the neuro evaluation outcomes, and off he went.

PT Clinical Decision-Making
Is this an emergency? Yes, single patient – needs evaluation of acute neurological changes, FAST criteria met, SDxT positive
Does patient need to move? Yes, immediately
How does this patient need to move? Yes, lift team transfer to stretcher from floor and by ambulance to hospital


Although these tools are specific to screening for cerebrovascular accidents, the Medical Priority Dispatch System utilizes an algorithm to screen for several emergency medical events. You can take a look at more tools here:

They have emergency protocols for tourniquet use, emergency child delivery, and even active shooter response! There is a section on nurse triage, fire, and many others!


Your clinical knowledge as a Physical Therapist (or other Rehab Clinician) goes a long way, even when other tools can’t give you clear answers. Overall, even though both of these patients were assessed with the same tools, both of them had history of strokes, and both of them gave confounding outcomes, they BOTH required urgent medical care for one reason or another. Having both tools in your pocket gives a higher percentage shot of remembering how to identify the need for emergency response and having a baseline comparison was key for both patients to identify the level of need. Eventually, it was clinical knowledge that determined the cause (not mine, but some very smart provider at a hospital), but it was the initial tools that got the patients the care they needed in time.


Have you used the SDxT or the FAST tool? I’d love to hear your story in the comments!

It’s Getting Hot in Here: Body Temperature

How many times have you had your temperature taken lately? I think I’ve had my temperature taken thousands of times in the last few months. We are seeing the increased use of forehead scanning thermometers and temporal scanners, all the non-contact forms of temperature assessment, to screen folks for COVID-19 symptoms upon entry to anyContinue reading “It’s Getting Hot in Here: Body Temperature”

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References:

Abegaz, T. M., Shehab, A., Gebreyohannes, E. A., Bhagavathula, A. S., & Elnour, A. A. (2017). Nonadherence to antihypertensive drugs: A systematic review and meta-analysis. Medicine96(4), e5641. https://doi.org/10.1097/MD.0000000000005641

Barron, T. (2013). MAKE IT FAST. The Journal of Emergency Dispatch. Retrieved from https://iaedjournal.org/make-it-fast/

Brice, J., et. al. STAT 911: Stroke Assessment Tool for 9-1-1 Dispatchers. https://www.ahajournals.org/doi/full/10.1161/STROKEAHA.108.545574

Buck, B. H., Starkman, S., Eckstein, M., Kidwell, C. S., Haines, J., Huang, R., Colby, D., & Saver, J. L. (2009). Dispatcher recognition of stroke using the National Academy Medical Priority Dispatch System. Stroke40(6), 2027–2030. https://doi.org/10.1161/STROKEAHA.108.545574

Clawson, J. J., Scott, G., Gardett, I., Youngquist, S., Taillac, P., Fivaz, C., & Olola, C. (2016). Predictive Ability of an Emergency Medical Dispatch Stroke Diagnostic Tool in Identifying Hospital-Confirmed Strokes. Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association25(8), 2031–2042. https://doi.org/10.1016/j.jstrokecerebrovasdis.2016.04.021

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PT/INRs: Helping Patients Manage their Anticoagulation

So, who’s up for some bloodwork? Venipuncture, anyone? Finger prick, maybe? No..? Oh… Well, ok then. I’ll carry on. I know it may sound a bit crazy, but as a PT, I actually do some (very) minor blood work. I’m not trained as a nurse or phlebotomist or anything like that. Nope. Just some on-the-job-training on doing PT/INRs.

In this case, PT doesn’t stand for Physical Therapist like it usually does in things I write and across the medical spectrum. In this case, PT stands for prothrombin time (or “protime”) and INR stands for international normalized ratio. These factors combine to tell us many things about the effectiveness fo anticoagulant therapy. And, I’m sure you guessed it, they are important for us, as rehab clinicians, to do our best work for our patients. A normal INR in a patient who is not pharmacologically anticoagulated is 1.0. When we are pharmacologically anticoagulating, we want the INR to be between 2.0 and 3.0 and I have even been asked to go up to 3.5 for certain patients with multiple risk factors or who have mechanical heart valves. Average PT is 10-14 seconds.

IF you find that INR is greater than 5.0, you should NOT be performing any kind of therapeutic intervention and you should NOT be exercising the patient. Obviously, they will need to move enough to access care, and they should do so safely, maybe even with your help to prevent falls, but outside of that, they are inappropriate for therapy. If you find an INR higher than 6.0, the patients needs to get immediate medical attention as they are at an unsafe risk of bleeding. If you find the INR is too low, mobilization helps with blood flow which decreases clotting, so you should be working to mobilize the patient as much as you can in the absence of signs and sympyoms of DVT or PEs. However, you will still need to contact the physician as the patient may need adjustments in their anticoagulant dosage.


Doing this is actually really easy. We use a small meter called a CoaguCheck. Some patients have their own but I carried one regularly because I did so many of these. A small finger prick is done with clean technique, a capillary chip is inserted in to the machine, the blood is placed on the capillary chip, and a reading appears. There are several more pieces to this because the machines can be VERY finicky. The biggest issue is usually getting blood! I’ve put some serious time in to managing an arm from proximal to distal in an effort to “milk” blood to the finger to make sure I can get enough.

The best tip I ever received in performing this procedure is that you need enough blood to make it looks like a lady bug is sitting on the finger. This needs to be significantly more blood than what is required for blood glucose monitoring. I know that doesn’t sound like a lot, but if that blood is not therapeutically coagulated, it can be really difficult to get that much in the amount of time you are given. That’s the other hard part, the machine gives you a countdown in which you must apply the blood to the capillary chip. If you run the timer down, you have to start over with a new finger prick, new chip, and new reading.

Roche Diagnostics CoaguChek XS System Coaguchek XS System:Healthcare
Source

I typically go at this in a specific way each time: getting everything set up with clean technique, then get the chip, machine, and lancet ready. I then start my timer (which happens when you insert the capillary chip in to the meter) and spend about half the time (90 seconds) working the arm, hand, and finger to make sure I am going to get enough blood. Then I clean the finger, lance it, and “milk” enough blood out to look like a lady bug is sitting on the finger. Finally, I apply the capillary chip to the blood, allow capillary action to do its thing, and then wait for my reading.


Especially after a surgery, many of our patients are on prohpylactic anticoagulant therapies to reduce their risk of developing blood clots that could be potentially fatal if they travel throughout the circulatory system and relocate themselves in the lungs. But post-operative patients are not the only ones I help with this minor technique. As many of you know, I manage many patients with complex chronic diseases that require them to be heavily anticoagulated. These conditions may include heart failure with atrial fibrillation, cancer when chemo effects to the heart, or they have hypertension, diabetes, and Factor V Leiden deficiency. There are a million combinations I could list here. Regardless, most of them end up on Coumadin/Warfarin for anticoagulation which means their PT/INRs have to be monitored for therapeutic effect.

Now, of course there are other options for long term anticoagulation, such a rivaroxaban (Xarelto) or apixaban (Eliquis). These have some serious benefits like not having your finger pricked weekly to monitor your PT/INR, not having to worry about dietary choices (see below), and having fewer drug-to-drug interactions. However, when you have a patient with a history or high risk of falls, you don’t really want them on these particular options. Even though the effects wear off sooner, once the anticoagulant is in effect, it cannot be reversed. Once you start bleeding, you keep bleeding. That’s a real concern when you develop a subdural hematoma from a fall.

Photo by Karolina Grabowska on Pexels.com

Another drug in this “new” class of anticoagulants is Dabigatran (Pradaxa). I listed this one separately because a clotting antidote has actually been developed called idarucizumab (PraxBind). However, Pradaxa can still be hard on kidney function and tends to be incompatible with mechanical heart valves, similar to the other drugs in the “new” class of anticoagulants.


Things to watch out for when you have a patient utilizing Coumadin/Warfarin (outside of the obvious bruising/bleeding) include:

  • Medication Interactions. All generally administered medications included
    • Products containing acetaminophen
    • Most broad spectrum antibiotics
    • Aspirin
    • all other NSAIDs
    • Most antacids and laxatives
    • Antifungals
    • Amiodarone (or other rhythm drugs)
  • Supplements. Just don’t. There are many interactions with various supplements and they are not well controlled or easy to treat.
  • Foods. Anything high in Vitamin K. This means all leafy green and dark purple vegetables and fruits. This also means green tea. Like I mentioned above, Vitamin K is the antidote to Coumadin bleeding, so eating foods with higher levels of vitamin K will decrease the effectiveness of the drug.
    • Other foods include grapefruit, cranberry, garlic, and black licorice
  • Alcohol. Alcohol is also a blood thinner so you don’t want to double down on the effects of the medications.

Also, be on the lookout for bleeding that isn’t obvious. This can be GI bleeding, subdermal bleeding, internal bleeding, or cranial bleeding which would result in:

black or coffee-grounds stool or vomit
bleeding of the gums when brushing teeth (not otherwise explained)
severe head aches or stomach aches
dizziness, weakness, fatigue
low HgB and low Hct on CBCs
shortness of breath with minimal activity or at rest
new onset or increase in falls
ecchymosis of an entire limb or quadrant

All of these things would warrant immediate evaluation by a medical physician, probably in urgent care or emergency depending on the severity. Patients may need to receive the Coumadin antidote, Vitamin K or PCC.


Photo by Daria Shevtsova on Pexels.com

Other considerations include the need for dietary support. Call in a consult to your dietician because these folks aren’t going to be able to eat much from a large class of foods that would otherwise be REALLY good for them. It’s not that people who take Coumadin/Warfarin CAN’T have foods with vitamin K, but they have to eat about the same amount every day to keep the blood levels steady. Spikes in the amount of Vitamin K people eat are what causes problems with their anticoagulation therapy. And, given we are basically asking people to limit a large category of foods that are otherwise REALLY healthy, people who take Coumadin/Warfarin are going to need some serious dietary support to avoid adding obesity to their list of medical conditions.


So, who’s up for picking some fingers? It’s a great easy may to help patient’s manage their chronic conditions in the home, on the go, or in a short amount of time. Expanding your role and fulfilling your scope is only going to help your patients be their best selves and make their best choices.


How about you? Do you do anything in your practice that is something you learned on the job but not otherwise part of your standard professional practice? Tell me about it in the comments!

More Reads on Chronic Disease Management…

Dehydration

Gosh, this is a fine line… Especially in the patients I regularly see. A colleague and I often say, “If you have any more water, you’ll die. If you don’t have any more water, you’ll die.” This is actually a frequent education topic that I cover with patients. Scary? Yes, but true. So, why isContinue reading “Dehydration”

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References:

Collins, S. & Beckerman, J. (2016). How Do New Blood Thinners Compare to Warfarin? Retrieved from https://www.webmd.com/heart-disease/atrial-fibrillation/features/warfarin-new-blood-thinners

Mayo Clinic. (2020). Prothrombin time test. Retrieved from https://www.mayoclinic.org/tests-procedures/prothrombin-time/about/pac-20384661#:~:text=In%20healthy%20people%20an%20INR,in%20the%20leg%20or%20lung.

Wax, E., Zieve, D., Conaway, B. (2019). Vitamin K. Medline. Retrieved from https://medlineplus.gov/ency/article/002407.htm

Follow @DoctorBthePT on Twitter for regular updates!

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It’s Getting Hot in Here: Body Temperature

How many times have you had your temperature taken lately? I think I’ve had my temperature taken thousands of times in the last few months. We are seeing the increased use of forehead scanning thermometers and temporal scanners, all the non-contact forms of temperature assessment, to screen folks for COVID-19 symptoms upon entry to any public space. Are you taking temperatures in your PT clinic? I hope so. It’s not a perfect assessment, but it’s better than nothing and at least gives the appearance of attempting to create a safe patient environment.

There’s a few considerations for temperature that can be really relevant to PT practice outside of just making sure your patients don’t have COVID-19. That’s right, ya’ll! We are going back to our old friend, Sepsis. We talked about sepsis in quite a few posts in the past, and it keep rearing it’s super ugly head in so many topics. You may be thinking, “Doctor B, I really don’t see that many patients with sepsis, so I’m not sure where you are going here…” I may be the odd one out, but I’ve seen too many patients with sepsis. Even one is too many, but I’ve seen far too many. I wouldn’t challenge you that, depending on your setting, you’ve probably seen more patients with sepsis than you realize. If you are in general acute care, skilled nursing, or home care, sepsis is everywhere, but it takes on different names, usually the names of the infectious entities that cause it.

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I’m sure you’ve heard of all the infections that land people in the hospital for “pneumonia”, clostridium difficile (c. diff), MRSA, VRSA, “staph”, etc…… All of these land people in the hospital because they have, to some point, gone septic, meaning the pathogens have entered the blood stream (aka bacteremia, septicemia, blood poisoning) making them far more difficult to fight off without help. But, as PT’s how do we detect sepsis? If you read the headliner on this article, you should probably have an idea of where I’m going.

Detecting sepsis and other infections is a top priority job for PTs and other rehab professionals. He look for signs and symptoms of infection all the time, from redness and swelling to exudate and smell. But infections can be so much more subtle than that. And sepsis can be hidden.

The Basics

Remember that human beings can only function properly in a certain homeostatic environment. Just like our pH range is so small (7.35-7.45 ideal range), our temperature range is also pretty narrow: 96.0 F to 100.4 F. Obviously, that doesn’t mean we can’t somewhat function outside of these parameters, but the function is not ideal.

That’s right, normal temperature is a range. It’s not just 98.6 F. That’s because “normal” depends on where an dhow the reading was taken. Temperature can be taken at any of the following locations, but be sure to document the location as there are different “normal” temperatures for each location.

  • Oral/Sublingual
  • Temporal
  • Ear
  • Axillary
  • Forehead
  • Rectal
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Not to say that any one is better than the other as they all have their clinical place, but some are more accurate. However, it may be significantly difficult to get an accurate oral temperature on a screaming infant. And most adults will shy away from a rectal thermometer. So, even though they differ in accuracy, they can all be useful. Clearly, even though forehead scanning is one of the least accurate, it has its place in the new “contact-less” environment in which we function.

Hypothermia

So, what if a body temperature is below 96.0 F? Remember that body temperature elevates to ward off invaders. It is part of our body’s immune response. If the temperature doesn’t respond to invaders, the body is NOT fighting. Sometimes, the temperature can even decrease. I have actually had a patient where a sublingual temperature of 95.2 F was his only symptom of sepsis because his body was NOT fighting the infection and we caught it early. A decline in body temperature can happen for a number of reasons:

  • Advanced age resulting in less efficient immune function
  • Multiple infections overwhelming the immune system
  • Overload of antipyretic medications (acetaminophen, ibuprofen, some antihypertensives…)
  • Certain genetic conditions (Prader-Willi Syndrome)

Consequently, the list ofd things that makes someone more susceptible to septic infections looks pretty similar with the addition of:

Hyperthermia

AKA Fever. However, hyperthermia can happen for reasons other than infection such as with heat stroke. This is also something we need to be monitoring in our patients as we may be asking them to physically exert themselves, whether in our presence or not, through exercise on a hot day. We may also see patients who have a host of thermoregulation conditions that can be caused by:

Remember that an elevation in body temperature is a response to an infectious agent of some kind. Invaders tend to only be able to live in certain environments so the body elevates the temperature to change the environment and make it unfavorable or uninhabitable for the invader. Elevation of body temperature can also happen in the evenings as your body begins its “clean-up” process. However, most medical professionals don’t consider elevated body temperature to be febrile until it is 100.5 F or greater.

Clinical Application

If you are taking a full set of vitals anyway, blood pressure, heart rate, oxygen saturation, you may as well take a temperature, too. Especially right now, temperature matters and is important to our fully informed treatment of any patient. However, if you have someone who is post-operative of ANY KIND, you should be taking their temperature at your visits. We are the front line against infections in our post-operative patients.

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And in our treatment of populations on either end of the age spectrum, we need to constantly be putting sepsis on the list when we consider our differential diagnosis. I’m not saying it needs to be front and center, butI’m not saying it shouldn’t be, either, especially given the pandemic climate. This also then feeds in to the ongoing screening of people for COVID-19 and other infections that we discussed HERE.


How many COVID-19 screenings have you done in the past week? Tell me in the comments!

Sternal Precautions

“Patients exchanging habits of activity for complete rest are likely to become rapidly worse.” This quote fuels my everyday. These words have informed nonsurgical and surgical rehabilitation and its evolution from handing out bed rest like Oprah hands out cars to getting people moving early and keeping them moving often. Here’s the crazy thing: ThisContinue reading “Sternal Precautions”

Diaphragmatic Breathing

Let’s talk about this super simple technique that can change everything. Diaphragmatic breathing is really just how we are all supposed to be breathing most of the time. The purpose of the diaphragm is to facilitate breathing. Diaphragmatic breathing improves gas exchange and increases lung volumes. These are all really good things if we need toContinue reading “Diaphragmatic Breathing”

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References

CDC. (2020). Sepsis. Retrieved from https://www.cdc.gov/sepsis/what-is-sepsis.html

HealthWise Staff. (2019). Fever Temperatures: Accuracy and Comparison. C.S. Mott Children’s Hospital, University of Michigan Medicine. Retrieved from https://www.mottchildren.org/health-library/tw9223

Skelton, F. (2016). Rehabilitation of Central Nervous System Disorders: Impaired Thermoregulation. American Association of Physical Medicine and Rehabilitation. Retrieved from https://now.aapmr.org/impaired-thermoregulation/

Follow @DoctorBthePT on Twitter for regular updates!

COVID-19: Where the Heck Are We?

I started this venture back in March in response to COVID-19. I saw many Physical Therapists and other rehab professionals shutting their doors because it wasn’t safe to keep them open. I saw an opportunity to improve our profession by addressing a deficit in practice: the lack of cardiopulmonary skill implementation. It wasn’t even that we didn’t have the skills, because we absolutely do! We learn all this stuff in school and sometimes its just a matter of how much you practice it. Implementation has always been my concern. We have tons of research saying all the best things about exercise, but we don’t implement most of it. Implementation is key. So, I was really just hoping to help people brush up on things they already knew or apply them in different ways so they could keep working and keep practicing even in the midst of a global pandemic.

Along the way, this process became complicated because what we all knew as fact was changing. The view of “science” changed. Lots of things changed. The organizations we thought we could rely on failed us. And, to the entire medical world still practicing, we felt like every day, sometimes every hour, was a slap in the face. COVID-19 sure has made an impact on the world, but I can’t even begin to explain the impact it’s had on the medical community.

So, where does that leave us? COVID-19 is still here and we are still treating it, so let’s take a look at some data on where we are at in this thing.


All the Numbers and Changes Over Time

I hate this part. There are a lot of different data sources to use here. All of them will be different due to different reporting metrics and time frames. So don’t be surprised if all the numbers aren’t EXACTLY the same. There are also different ways to combine, separate, and read the data, which also creates differences in numbers. We are going to go with the data from Johns Hopkins University of Medicine. Why? Because they run one of the largest live updated databases of this information that draws from several other databases. Say what you want, I’m going with the largest amount of information aggregated by the leading professionals.

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At the time of writing this: Global Cases are at 24,563,393. The US has over one fifth of those at 5,901,393. Global deaths are at 833,466. The US has contributed 181,409 of those. Global recovered cases are 16,051,454. The US has contributed 2,101,326 of the recoveries. Total tests performed in the US is at 75,301,306. And the number of hospitalized patients in the US is 365,993.

Lots of number, I know. But we are just going to do some simple things with them. And, for some reason, people seem to want the numbers of COVID-19. They have never mattered before when millions were dying of flu or tuberculosis or malaria but whatever…

These numbers tell us that there are still 7,678,473 people in the world actively suffering from COVID-19 (number of total cases – recoveries – deaths = ongoing cases). This may not include the long-haulers which are usually viewed as “recovered” even though they would sorely disagree. In the US, that leaves 3,618,658 people (about half of the world total) still fighting COVID-19, or just over 1% of the population of the US. I know 1% doesn’t sound like much, but when you know that 1% is more than three and a half million people, that 1% looks pretty big to me.

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We also need to understanding that all of these numbers are an underestimation of the actual picture. We can’t count any cases that happened before testing because we didn’t have testing to prove there were positive cases! So all the cases that happened back in February and March (and some even earlier, researchers are now finding) didn’t make the count. That includes positive cases, deaths, hospitalizations, and recoveries. Doctors at that time were treating COVID-19 based on severity of symptoms, imaging studies, and based on exclusion criteria. We also need to remember that testing has been pretty restricted in the grand scheme. Even if you’ve had an exposure, you can’t be test until you show symptoms, which doesn’t prevent you from spreading it around for a few days first. And, for a while, there were so few tests that only those at highest risk were every tested. So, many cases went unreported.

That’s a lot of numbers… Glad that’s over…


What else is going on?

Symptoms of COVID-19 can range from all the typical symptoms of a respiratory virus (coughing, fever, shortness of breath) to GI symptoms (nausea, vomitting, dehydration) to strange presentations like wounds on the toes. Neurological symptoms are well know to be involved such as confusion, loss of smell or taste sensations, or a headache. Serious presentations may include blood clots, strokes, heart attacks, acute respiratory distress, or altered mental status. It all depends on where the infection takes hold in your body and what the mechanism of entry was. Speaking of how someone gets infected…

We are still experiencing a PPE shortage. And providers who experienced a PPE shortage are more likely to have been infected with COVID-19. N95s are still the most frequently reported shortage item, but even gloves are beginning to be in short supply. Even with the PPE shortage, though, health care providers who wore a mask at every clinical interaction were less likely to contract COVID-19. Healthcare Providers still rank among the highest risk people to contract COVID-19.

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Although the CDC maintains its stance on COVID-19 only being transmitted through droplets (aka droplet precautions), many research studies have determined that COVID-19 is airborne. The WHO has also acknowledged that there is significant evidence to support the likelihood of airborne transmission. Even many employers have acknowledged that their providers at at an increased risk and provided the supplies necessary for airborne precautions. As if things weren’t already confusing enough, even though the CDC maintains their stance on droplet precautions, they recommend airborne precautions when performing aerosol-generating procedures. Thankfully, many professional organizations have come forth to define “aerosol-generating procedure” to help guide treatment safety.

A large field of research has been devoted to determining the ventilation and air purification needs that rooms of different sizes and capacities would require. Healthcare engineering in many capacities, from 3D printing shields and N95s to improving sanitization to HVAC updates, have been working overtime to fuel the needs of providers and patients.

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Long haulers have emerged. These are the folks, whether hospitalized for COVID-19 or not, that continue to experience symptoms of COVID-19 long after the standard infection and illness window. Some people who believe they were infected back in February before testing was even available, are still experiencing the fatigue, shortness of breath, and muscle aches that resulted from their infections.


Have we made any progress? What are we doing about this?

The medical world and the non-medical world have started to blend together, for better and for worse. Post-Intensive Care Syndrome (PICS) has been making headlines and the therapy world (including respiratory therapists) are getting some serious spotlight. The world has become aware of the fact that people don’t just jump back from serious diseases. Proning, an intervention that has been around for a long time, has also been given screen time. These particular topics have placed rehabilitation front and center. Considering most people don’t even know what PTs and OTs do, and think SLPs just play mind games all day, I’d say we’ve made some positive public image impact.

Some professional organizations are also stepping up and providing toolkits, new tools, and educational content regarding treating patients with COVID-19. There is also guidance issued regarding screening for COVID-19 in the outpatient setting to decrease transmission and infection risk.

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Like the numbers above have shown, over 2.1 million people have recovered and therapy played a pretty big role in helping some of those people recover. The early numbers from back in May showed that about 20% of the people who contracted COVID-19 would require extra medical help to recover, and about 10% (half of the 20%) would need long term inpatient care. We’ve gone from dreading the intubation and ventilation phase to seeing many of these people in the home and outpatient settings for treatment, management, and recovery. Outpatient clinics have reopened and begun utilizing technology to help more people than ever!

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Telehealth is the biggest win to come from COVID-19. The implementation of telemedicine into the therapy world used to be a pipedream. It was only for expensive, private-pay therapy. Today, most payers reimburse for telehealth visits to some extent. We can now provide follow ups, evaluations, check ups and check ins, virtual home programs, and so many other services to our patients without have to risk their health or our own. If anything about COVID-19 sticks around for the better, I hope it’s telemedicine for the rehab world. Thankfully, CMS has indicated that they have no intention of repealing the telemedicine reimbursement program. That’s really great because these long haulers are needing long curses of therapy and many others who were hospitalized are needing frequent and long term rehabilitation for severe deconditioning and pulmonary function impairments.

Research has indicated that the suspected time frame that COVID-19 will significantly impact our lives in 18-24 months. The research on immunity from previous infections is mixed with some people showing antibodies long term and some people not showing any antibodies to the infection after a known case. Herd immunity has been discussed as a solution, but that can’t happen is antibodies don’t stick around enough. Not to mention it would require over 1,000,000 MORE deaths to achieve than what we currently have. Not quite an acceptable solution. Vaccines are all in the works from multiple different companies in a race to the cure. From a rehab perspective, we need to be screening every patient. We have the abilities, skills, and knowledge to do so. You can read more about that here.


Overall, our world has completely changed, at home and at work. COVID-19 doesn’t look like it’s going away anytime soon. The SARS-CoV-2 virus has definitely mutated over time with more than 6 different strains already detected. The acuity of illness seems to be decreasing, but the symptoms seem to be lasting longer. It will probably continue to do so. I hope this somewhat brings you up to speed to where we are now. I’ll keep updating you on big changes. And I’ll keep helping you update your skills to best treat your patients, COVID-19 or not.


Are you still experiencing PPE shortages at your workplace? Tell me about it in the comments!

I’ll Huff and I’ll Puff… But the Huff Will Be More Effective

What is a huff? Huffing is one of the more difficulty techniques to learn for airway clearance. I find that most of my patients have trouble mustering the strength to perform a good solid huff. It’s not just that it is unfamiliar, but also that it takes diaphragm strength that they just don’t have. ButContinue reading “I’ll Huff and I’ll Puff… But the Huff Will Be More Effective”

Vibration & Percussion

If you read my post on airway clearance techniques, you probably saw vibration and percussion down at the bottom of the force progression. Although I have covered several other pieces of the force progression (Active Cycle of Breathing, PEP Devices, etc) in subsequent posts, I haven’t touched on these topics yet because they haven’t beenContinue reading “Vibration & Percussion”

More Than Just A Respiratory Disease: The Tools You Need to Rehab COVID-19

Isn’t COVID-19 just a respiratory disease? If only that was true. We are good at treating respiratory infections. We have lots of drugs for viral, bacterial, parasitic, and fungal infections of the lungs. Most of them work really well! We also have several back-up treatments, inhaled medications, and adjuvant therapies (like rehab!) that make primaryContinue reading “More Than Just A Respiratory Disease: The Tools You Need to Rehab COVID-19”

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Resources:

CDC (Updated August 12, 2020). COVID-19 Overview and Infection Prevention and Control Priorities in non-US Healthcare Settings. https://www.cdc.gov/coronavirus/2019-ncov/hcp/non-us-settings/overview/index.html#:~:text=COVID%2D19%20is%20primarily%20transmitted,be%20inhaled%20into%20the%20lungs.

Dong, E., Du, H., Gardner, L. (2020). An interactive web-based dashboard to track COVID-19 in real time. The Lancet: Infectious Disease: CORRESPONDENCE 20(5):533-534. DOI:https://doi.org/10.1016/S1473-3099(20)30120-1PlumX Metrics.

COVID-19 Dashboard by the Center for Systems Science and Engineering (CSSE) at Johns Hopkins University (JHU). https://coronavirus.jhu.edu/map.html

Self WH, Tenforde MW, Stubblefield WB, et al. (2020). Seroprevalence of SARS-CoV-2 Among Frontline Health Care Personnel in a Multistate Hospital Network — 13 Academic Medical Centers, April–June 2020. MMWR Morb Mortal Wkly Rep. DOI: http://dx.doi.org/10.15585/mmwr.mm6935e2external icon

Other resources can be found on the linked pages to which they apply.

Follow @DoctorBthePT on Twitter for regular updates!

Sternal Precautions

“Patients exchanging habits of activity for complete rest are likely to become rapidly worse.”

This quote fuels my everyday. These words have informed nonsurgical and surgical rehabilitation and its evolution from handing out bed rest like Oprah hands out cars to getting people moving early and keeping them moving often. Here’s the crazy thing: This quote was said by Austin Flint in 1886 in specific reference to the rehabilitation of patients with heart disease. 134 years later, we are still fighting the battle against overly-restrictive precautions and rest orders across all disciplines and diagnoses.

I don’t know if you are aware of how a sternotomy goes down, but it’s pretty aggressive. There is a lot of tissue stretching, stabbing (yes, literally), cutting, burning, and wiring. And then there’s the retractors. Those babies get in that tiny space and make it so much larger! No wonder patients are sore afterward. You can go watch a video on YouTube to see what it’s all about. Patients typically have discomfort in their thoracic and cervical spines, shoulders, ribs, anterior superior chest and joint articulations… and that’s just for a standard valve replacement! If you add a CABG, we can add peripheral wounds in the arms and legs, and if we are talking organ replacement, the back of the head, low back, and hips can get pretty sore from positioning and the use of a bump to help open the rib cage.

Even though the first coronary artery bypass graft (CABG) wasn’t performed for the treatment of heart disease until 1960, infection and dehiscence of the sternotomoy were relatively common (not terribly surprising considering what goes in to it!). Of those who experienced these complications, up to 50% died. Needless to say, surgeons felt like they needed to get extra cautious. However, there was never any research performed to discern which movements of the body or extremities stressed the sternum the most, and whether or not this stress impaired healing. Thus, sternal precautions were laid down.


There is NO set standard for sternal precautions

Yup. Different surgeons, medical centers, and states all do it differently. There is also no set standard for when or how to reduce or remove sternal precautions. In the heart and lung transplant arena, our surgeons and facilities tended to decrease sternal precautions over time, allowing one or two upper extremity movements every month or so and decreasing the weight lifting restriction by 5-10 pounds every month or so. Real consistent, right? For further evidence of these inconsistencies, check out this table which shows you three different sets of “sternal precautions” from three different major medical centers.

source

And to complicate matters even further, many post-operative exercise prescriptions often involve a standard set of exercises, many of which violate one or several sternal precautions that are initially given! I’ve found this to be true at more than one location! One survey performed in 2011 found more than 28 different versions of sternal precautions! And, to add one more level of complication, Physical Therapists and Cardiothoracic Surgeons do sternal precautions very differently.

Top 5 sternal precautions prescribed by cardiothoracic surgeons:
  1. Lifting no more than 10 pounds of weight bilaterally
  2. Lifting no more than 10 pounds of weight unilaterally
  3. Bilateral sports restrictions
  4. No driving
  5. Unilateral sports restrictions
Top 5 sternal precautions reported by physical therapists in order of importance:
  1. Lifting no more than 10 pounds of weight bilaterally
  2. No hand over head activities bilaterally
  3. Bilateral sports restrictions
  4. No driving
  5. Active bilateral shoulder flexion no greater than 90°
And ACSM tells us this:

For 5 to 8 weeks after cardiothoracic surgery, lifting with the upper extremities should be restricted to 5 to 8 pounds (2.27-3.63 kg). Range of motion (ROM) exercises and lifting 1 to 3 pounds (0.45-1.36 kg) with the arms is permissible if there is no evidence of sternal instability, as detected by movement in the sternum, pain, cracking, or popping. Patients should be advised to limit ROM within the onset of feelings of pulling on the incision or mild pain.


There has been some limited research that showed that pushing up from a chair during sit to stand activities provided the greatest physical stress force to the sternum. However, these patients had known chronic sternal instabilities, so the cases are slightly different. Even though the stress has been measured to be physically less, unilateral loaded arm movements caused the greatest pain in these patients. Clinically, I have seen this to be the case. Unilateral upper extremity loading, such as when laying on the side or reaching for a cup of coffee, always seems like the most painful things.

Photo by Gustavo Fring on Pexels.com

Take notice, there is nothing in any of these discussion on sternal precautions that mentions splinting the sternum. For as many years as I remember, including before I was a therapist, we have used nice heart or lung shaped pillows to splint the sternum during coughing or sneezing. If the pillow wasn’t available, we taught patients to splint their sternum with their hands or a different pillow. I even took to educating patients about their first sneeze and being ready. That stuff doesn’t show up anywhere in here.

Speaking of a sneeze, there was a study published in Thoracic Cardiovascular Surgery that stated the force across the sternum with a single cough is greater than that measured lifting 40-pound weights. The authors had serious concerns about the lack of surgeon confidence in their owns repairs if the sternotomy couldn’t support 5 pounds of force.

And, we need to talk about the biggest elephant in the room…

Driving. Everyone’s first question after they get home is, “When can I drive again?” There is absolutely no standard recommendation for this one, either. Some physicians say 3-4 weeks once they’ve had their follow-up visit, whereas some say 6-8 weeks. Some patients have been told to lay in the back seat or not wear a seatbelt when they are traveling in a car! These are just generally not safe recommendations from any provider, considering there is a long history of research telling us that wearing a seatbelt and using an airbag decreases the risk of a driver’s sternum contacting the steering wheel. Are we concerned about the patient being in an accident? Sure! But you can’t control everything. The research shows us that there is no higher risk of sternal fracture after a healed sternotomy than if there was no sternotomy.

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So what do we actually know?

What we know from the most recent research is that sternal precautions don’t matter. There, I said it. I spent years teaching other therapists about sternal precautions, all the while thinking, “why do we do this to ourselves and our patients?”

What actually matters is allowing patients’ pain or discomfort at their sternum to guide their movements. A randomized multi-center single blinded study showed that there was absolutely no difference in physical, kinesphobic, pain, or quality of life outcomes and no increased rate of complications when patients were simply asked to just let their post-operative discomfort guide their movement.

“There is no universally accepted definition causing application of SP [sternal precautions] to be largely arbitrary.”

Cahalin, et al. 2011

As far as driving goes, after sternotomy, only minimal forces on the sternum were found with the activities of driving. We actually have found that the safest place in the car for the patient to be is in a seat with an airbag and wearing a seat belt. Whether that is a front passenger or a driver is unclear. Not only does the ability to drive have a huge impact on quality of life, but the lack of driving ability restricts patients from participating in cardiac rehabilitation programs because they can’t get there!

Now, of course, there are wonderful home care therapists to help with this, but they are not always available in all areas and many can’t provide standardized sub-maximal and maximal exercise tests because those require a treadmill or exercise bike and EKG monitoring. So, patients end up having to sit at home, not rehabbing for up to 8 weeks. That’s 8 weeks that they are not only losing muscle mass, aerobic capacity, and functional ability, but 8 weeks that they are NOT working toward improving any of those things like they could in Cardiac Rehab! That’s 16 total weeks of lost time in the recovery from one procedure, simply because the patient couldn’t drive.

Photo by Andrea Piacquadio on Pexels.com

Of course, not all patients are appropriate to drive after having an open-heart related procedure, but evidence shows us that if they are tolerating their medications well and aren’t having persistent arrhythmias, they should be cleared to drive as soon as they are able. I will say, though, that having seen many patients at home for a PT evaluation after open-heart surgical procedures of all kinds, I’m typically pretty glad we got to do a home-based session. Many of them need education on positioning for sleep, infection control, and activity guidelines. You all know by now that I’m a pusher so I always work these people pretty hard. But, you know what? They all got better and there were never any negative outcomes associated with exercise or activity participation. Darn… Should have done a retrospective study on that one…

So what’s the answer? Some authors have suggested a graded approach to precautions instead of the standard blanket precautions laid down on everyone for the same procedures. Some authors have suggested letting discomfort be their guide and allowing any activities and movements that are not placing the patient at a significantly increased risk of nonunion. Some authors have suggested only giving some kind of patient-specific sternal precautions those patients who are already at higher risk of nonunion due to other comorbidities (such as diabetes or osteoporosis).

Overall, most of the rehab-focused research on sternal precautions has found that they are overly-cautious, restrictive instead of cautionary, and impede physical rehabilitation, which we know is so desperately needed by many after these procedures.


What are the sternal precautions at your facility? Do different doctors give you different precautions? Tell me more in the comments!

A Public Service Announcement: The Chain of Infection (and How YOU Can Break It!)

This post is written for one and all. If you are not a rehabilitation or medical professional, please read this post. Even if you are, please read this post. I’m going to address some things that need clarification. You can have all the opinions you would like, but there are some things that are justContinue reading “A Public Service Announcement: The Chain of Infection (and How YOU Can Break It!)”

Open Heart, Open Mind… Learning About Amio-Loading

I’m always learning something new. I called the cardiologist after an evaluation to report some severe orthostatic hypotension and the nurse and I got to talking. She was going back through the patient’s history and looking for why this may be happening. I had just finished assessing the patient in their home and they wereContinue reading “Open Heart, Open Mind… Learning About Amio-Loading”

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Resources:

Balady, G.J., Ades, P.A., Bittner, V.A., Franklin, B.A., Gordon, N.F., Thomas, R.J., Tomaselli, G.F., Yancy, C.W., American Heart Association Science Advisory and Coordinating Committee. (2011). Referral, enrollment, and delivery of cardiac rehabilitation/secondary prevention programs at clinical centers and beyond: a presidential advisory from the American Heart Association. Circulation. 124(25):2951-60.

Cahalin, L. P., Lapier, T. K., & Shaw, D. K. (2011). Sternal Precautions: Is It Time for Change? Precautions versus Restrictions – A Review of Literature and Recommendations for Revision. Cardiopulmonary physical therapy journal22(1), 5–15.

Crabtree TD, Codd JE, Fraser VJ, Bailey MS, Olsen MA, Damiano RJ. Multivariate analysis of risk factors for deep and superficial sternal infection after coronary artery bypass grafting at a tertiary care medical center. Sem Thorac Cardiovasc Surg. 2004;16:53–61.

El-Ansary D, Waddington G, Adams R. Relationship between pain and upper limb movement in patients with chronic sternal instability following cardiac surgery. Physiother Theory Prac. 2007;23(5):273–280.

Gach, R., Triano, S., El-Ansary, D., Parker, R., & Adams, J. (2019). Altering driving restrictions after median sternotomy. Proceedings (Baylor University. Medical Center)32(2), 301–302. https://doi.org/10.1080/08998280.2018.1551298

Katijjahbe, M. A., Granger, C. L., Denehy, L., Royse, A., Royse, C., Bates, R., Logie, S., Nur Ayub, M. A., Clarke, S., & El-Ansary, D. (2018). Standard restrictive sternal precautions and modified sternal precautions had similar effects in people after cardiac surgery via median sternotomy (‘SMART’ Trial): a randomised trial. Journal of physiotherapy64(2), 97–106. https://doi.org/10.1016/j.jphys.2018.02.013

Knobloch, K., Wagner, S., Haasper, C., Probst, C., Krettek, C., Otte, D., Richter, M. (2006). Sternal fractures occur most often in old cars to seat-belted drivers without any airbag often with concomitant spinal injuries: clinical findings and technical collision variables among 42,055 crash victims. Ann Thorac Surg. 82(2):444-50.

McGregor, W.E., Trumble, D.R., Magovern, J. A. (1999). Mechanical analysis of midline sternotomy wound closure. J Thorac Cardiovasc Surg. 117(6):1144-50.

Parker, R., Adams, J.L., Ogalo, G., et al. (2008) Current activity guidelines for CABG patients are too restrictive: a comparison of the forces exerted on the median sternotomy during a cough vs. lifting activities combined with valsalva maneuver. Thorac Cardiovasc Surg. 56(4):190–194.

Pratt, J.H. (1920) Rest and exercise in the treatment of heart disease. South Med J. 13:481–485.

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Dehydration

Gosh, this is a fine line… Especially in the patients I regularly see. A colleague and I often say, “If you have any more water, you’ll die. If you don’t have any more water, you’ll die.” This is actually a frequent education topic that I cover with patients. Scary? Yes, but true. So, why is a Physical Therapist going to talk to you about dehydration and water intake? I cannot even begin to explain to you in any short way how important this is to my DAILY practice.

Symptoms of dehydration include:

  • dizziness
  • light headedness
  • low blood pressure
  • orthostatic hypotension
  • increased thirst
  • fatigue
  • confusion
  • falls
  • urinary tract infections
  • incontinence
  • lack of sweating (in severe cases)
  • loss of consciousness (in severe cases)
  • cardiac arrhythmias (in severe or chronic cases)
  • loss of thirst (in severe cases)
  • the list goes on…

I treat all of those things as symptoms of other conditions and some in isolation. I bet you do, too!

Acute, chronic, or severe dehydration results in hypovolemia, which means that your body doesn’t have enough volume (blood volume) to support normal function. Guess what, folks? Blood pressure is a function of blood volume. So, if you lose blood volume, you will also lose blood pressure. When you lose blood pressure, your heart rate has to increase to make up for the loss in cardiac output to keep you going. This results in vitals findings that include a low blood pressure and an elevated heart rate. This is part of the discussion around hemodynamic stability, one of my favorite things to talk about.

For example, I’ve had a patient just recently that demonstrated blood pressures at rest 96/54 mmHg and a heart rate of 104 bpm. Sometimes, you don’t see the change in blood pressure and heart rate at rest, but you may see it with activity. The other day, I was teaching an exercise test and our model had resting blood pressure of 120/86 mmHg and a heart rate of 70 bpm. With ONE MINUTE of marching in place, her blood pressure was 130/90 and her heart rate was 50 bpm. She was also having cardiac arrhythmias that were not present at rest. It took ONE MINUTE. That’s it. And, just an FYI, this was NOT a patient. This was your standard run of the mill adult walking around doing their normal thing. This adult could be walking in to your clinic at any moment.


Why, you might ask, are so many of Doctor B’s patients dehydrated? Well, there are lots of reasons. I’m going to use this post to segue into a few other topics, but the gist of it is that they are sick. And when you are as sick as they are, drinking enough water is either difficult or not indicated. That’s right. You read that correctly. For some of my patients, drinking too much water is actually exactly their problem. You can read more about that here. But the REAL REASON that many of my patients are dehydrated is because I check for it. Simply because I check for it, I’ve significantly increased the number of dehydrated adults on my caseload. Just because someone is sick doesn’t mean they are dehydrated, but if you never check, you’ll never know.

Many of my patients have difficulty breathing. Many of them have COPD or some other form of lung disease. If it’s difficult to breathe at rest, getting up to get a drink of water is going to be pretty challenging, so many of them just don’t. For people who use supplemental oxygen, staying hydrated is even more important because their supplemental oxygen is very dry, even when using a humidifier, and causes cracks and bleeding in their nose. This causes discomfort and decreases compliance with their oxygen.

Photo by Daria Shevtsova on Pexels.com

If you’ve read my posts on heart failure, including the post that covers the Rule of 2s in relationship to heart failure, you can understand how too much water can actually become a problem. For patients who manage chronic heart failure, their fluid restriction (typically of 2 liters of water per day) can make them fairly dry, especially in the beginning of their disease. They are used to drinking as much water as they need, but all of a sudden, we are telling them they have to ration it out over the course of the day. Initially, they have to adjust to this change so they have some time frame of mild orthostasis or other dehydration symptoms. Funnily enough, they usually do fine for a while, but then they get too good at limiting their fluid intake and they start to REALLY limit themselves. Some patients are proud to announce that they can only drink 1 liter of water per day… And Doctor B cringes… Even when they have a fluid restriction, it is still important to drink to the max of their restriction to ensure they stay hydrated enough to function.

Many of my patients are also incontinent. We will talk more about this later, but the solution many of them think they’ve discovered is that, if they drink less water, they won’t have to get up to use the bathroom as often (which is hard for them to do, or requires the assistance of another person and they don’t want to be a bother). Unfortunately, this results in these older, more fragile people becoming dehydrated which produces hypovolemia, which results in increased sodium and potassium concentrations in their body (hypernatremia/hyperkalemia), which results in altered mental status and/or heart problems. Unfortunately for them, drinking less water doesn’t actually reduce their urination frequency….

That’s right, decreased water intake doesn’t really decrease your urine frequency. What actually happens is that your urine becomes more concentrated in your bladder. This concentrated urine becomes a bladder irritant which actually increases the frequency of feeling the need to go! This is especially true of those middle of the night trips to the bathroom that we all know result in falls. People stop drinking water and hour or two before bedtime so they won’t have to get up to use the bathroom in the night. But, darn it, that’s just not how it works. Now they will actually be getting up more often to use the bathroom because they are getting dehydrated before bed.

Photo by Pixabay on Pexels.com

Let’s factor in how often urinary incontinence co-occurs with chronic diseases, especially those of the respiratory system. You don’t have to go out and do your own research, I’ll just tell you: It’s pretty much every time. My patients often have to cough to clear secretions, because their throat is too dry, or because they have swallowing impairments. When they cough they become incontinent. They have trouble getting up so they just don’t. Then, they sit in that extra-concentrated urine until they muster the energy or someone is available to help them. They end up developing urinary tract infections which take them down the long spiraling road of confusion, falls, injuries, and hospitalizations.


Setting doesn’t matter. Many of my chronic disease folks are in the home health setting, but I saw plenty of dehydration in outpatient orthopedics, too. Even in the Skilled Nursing Facility where the nurses do daily rounds to check vitals, many more than once per day, I still check every patient before exercising them. Patients in facilities tend to be pretty dry, partially because they can’t get more to drink on their own. Finding patients too orthostatic to participate in therapy and recommending fluids has become a regular intervention.

I think you can all pretty much get the picture as to why not drinking enough water is really bad, especially for the older population or those with chronic diseases. And the threshold is pretty narrow for many people if they also have a fluid restriction. But, do you remember why I said so many of my patients have dehydration? The REAL reason? It’s because I check for it. And so should you.

Vitals. Every patient. Every time. More than once.

And you can be the one to prevent that hospitalization or that infection or that fall. All you have to do is look for it to know it’s there. Then you can intervene and be the hero with just a cup of water.


How many cups of water do you drink every day? Let me know in the comments!

Blood Pressure Basics

Are you taking the blood pressure and heart rate of EVERY patient you see for a new evaluation? How about for every visit? A recent survey of over 1800 Outpatient PTs showed that although 51% of PTs reported that over half their caseload had risk factors for hypertension and cardiovascular disease, only 14% of themContinue reading “Blood Pressure Basics”

ABGs (Part II)

So, now that you’ve read all the basics about ABGs in the first post, here is a little more about interpreting lab values and how to determine compensated conditions. This is where it gets fun! First off, let’s take a look at what NORMAL lab values probably look like: pH: 7.35-7.45 Partial pressure of oxygenContinue reading “ABGs (Part II)”

COVID Brain

Research is piling in regarding the neurological effects of COVID-19 and the depth of the research is giving us some really concrete information to help guide treatment, screening, and monitoring strategies. The anecdotal evidence is continuing to build quickly so I’ve significantly updated this post to reflect some of the more recent changes and findings.Continue reading “COVID Brain”

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Diaphragmatic Breathing

Let’s talk about this super simple technique that can change everything. Diaphragmatic breathing is really just how we are all supposed to be breathing most of the time. The purpose of the diaphragm is to facilitate breathing. Diaphragmatic breathing improves gas exchange and increases lung volumes. These are all really good things if we need to get more air in, such as in restrictive lung diseases like pneumonia, post-lung resection, or pulmonary fibrosis. It can also be a useful technique to mobilize secretions or other obstructions in the airway for people who have obstructive lung conditions like congestive heart failure or COPD.

Source

It can be a bit difficult to grasp the actions and movements of the diaphragm unless you understand its purpose. The diaphragm has a central tendon and attaches to the costal margins, spine, and xyphoid process to create a barrier between the thoracic cavity and abdominal cavity. There are openings in the diaphragm muscle (hiatus) for the passage of the major blood vessels (the descending aorta and inferior vena cava), the trachea, the esophagus, and a few other small circulatory structures.

As you can see in the picture, when this muscle is relaxed, all of its length is up inside the chest cavity. Per the standard skeletal muscle, when it contracts and the fibers shorten. This brings the insertion (the central tendon) to the origin (the costal margins, spine, xyphoid). The central tendon lowering down into the abdomen, increasing the volume of the thoracic space to allow for lung expansion and decreasing the volume of the abdominal space. There are other accommodations that happen to make up for this volumetric change, but we will talk about those in a different post.

Because of the negative pressure system within our pleural space, this increase in thoracic volume draws air into the lungs, allowing for increased ventilation and diffusion (aka gas exchange) to take place, specifically in the lower segments of the lungs. This allows for greater oxygen intake and great CO2 removal, or more efficient breathing, than if the diaphragm were not engaged during this task. Breathing that does not engage the diaphragm utilizes the upper intercostals and accessory muscles to open the upper portions of the lungs only for gas exchange. This is less efficient and is typically seen in patients who have chronic heart and lung diseases or spinal cord injuries.

Why do we stop breathing the way we should?

Well, there are many reasons. Skinny jeans, corsets, getting our picture taken, learned habits, sitting without a back support, spinal deformations, pain avoidance, central nervous system damage, gastrointestinal disturbances, space-occupying lesions, change in muscle strength or length (disuse atrophy or trigger points can come in to play here), changes in abdominal content volume (pregnancy or obesity), change in lung volume (resections), lung function changes (disease states)… the list is endless. The major factor is whether or not it becomes a persistent problem. The body is a smart system and it can compensate for pretty much anything, but, similar to other musculoskeletal impairments, long term compensations are what lead to problems.

Photo by freestocks.org on Pexels.com

What can we do about it?

Bring it back! The diaphragm may shut down for a number of reasons, but it can bounce back with pretty minimal input. The reason for that is because it is such an essential muscle! It utilizes three spinal levels to ensure its function (C-3, 4, 5, Stay Alive!) because redundancies ensure we can breathe, at least in part, even if there is some damage.

The reason we had to talk about all that old information about origins, insertions, and actions is because these things are going to be important when attempting to facilitate diaphragm function. We used to teach people to have patients lying in hooklying with an object of interest (TV remote, cell phone) on their abdomen. We would ask them to breath inward and use their air to raise the item on their abdomen. When the diaphragm contracts to compress the diaphragm, the contents have to go somewhere and that is usually outward, so the object rises. BUT HERE’S THE PROBLEM WITH THAT: The diaphragm doesn’t move that way! These techniques typically result in the patient performing a Valsalva maneuver.

The diaphragm doesn’t move up and down when in the hooklying position! It moved cranial/caudal, which is completely different. The diaphragm attaches all the way around our body (nearly 360 degrees), so we need to be facilitating it’s expansion all the way around, also! We can’t do that if someone is lying on their back for many reasons:

  • This applies resistance to the posterior segments of the diaphragm muscle, making it harder to activate all portions of the muscle.
  • The diaphragm needs to work in 360 functional space including all positions of the body! So diaphragm exercises need to be performed in sitting, standing, squatting, quadruped, laying, and whatever yoga pose you come up with next.

There is a small case for initially instructing diaphragm exercise in hooklying, because this allows for gravity neutral activation, but you better move on pretty quick if your patient is ambulatory and doesn’t have a high spinal cord injury, or you won’t be doing them any good.

Photo by Chevanon Photography on Pexels.com

What does diaphragm facilitation look like?

There are many ways to facilitate diaphragm activation but here are some I like to use:

  • Visual Feedback: Place the patient in a chair in front of a mirror and have them breathe in and out using their diaphragm. Do this with them so they can see what you look like as well.
  • Verbal cues:
    • “Breathe in deeply, bringing the air all the way down to the bottom of your lungs”
    • The “Triple Sniff”: Ask your patient to sniff quickly three times. The first two sniffs need to be short and the third one needs to be long.
  • Manual Facilitation: Place hands all the way around the patient! We cannot cue diaphragm breathing in one plane because that’s not how it happens so here are some options:
    • Basic: one hand on chest, one hand on abdomen – abdomen should raise equal to or more than chest with inhalation (not my favorite as it results in Valsalvas more times than not).
    • Better: Place C-shaped hands around the inferior costal margin bilaterally. Ask the the patient to breathe in to your hands.
    • Best: With your C-shaped hands, follow that inferior costal margin inward on the exhale and provide a deep quick stretch just before the inhale to cue diaphragm and lower intercostal muscle activation.

When utilizing manual facilitation, you will want to make sure you feel all margins of the insertion expanding and contracting so you may have to facilitate from several different points on the costal margin over several repetitions.

Any other tips, Doctor B?

The diaphragm works in concert with the pelvic floor. Make sure your patient doesn’t have to use the bathroom and isn’t tightening their pelvic floor again exhalation. This results in over-compression of the abdominal contents and result in prolapse or incontinence. This is especially a concern for female athletes, new mothers, and women who have chronic breathing problems. 55% of women without a chronic lung disease reported incontinence, whereas 71% of women with a chronic lung disease reported incontinence.

Female athletes (I’m looking at you, gymnasts!) can also use many other muscle systems to facilitate breathing and compensate for their diaphragm. Be sure to check out breathing with the scapulae and even the glutes! This can have serious implications for the pelvic floor, also.

Patients who have spinal cord injuries, or some degenerative neurological conditions such as MS, GBS, or ALS may have partially impaired diaphragm function and will require several different types of facilitation. Just like airway clearance techniques, these interventions are always best performed in addition to each other, not in isolation.


Patients who have chronic diseases that results in poor lung function, like we mentioned above, typically need to get more air OUT of their lungs before putting more air INTO their lungs with diaphragmatic breathing. Check out this post on dynamic hyperinflation to learn more about that. You may also find that facilitating diaphragmatic breathing results in expectoration of secretions for patients who have obstructive lung conditions. This is because they are finally getting some ventilation to these no-often-used parts of their lungs! Alternating techniques that remedy dynamic hyperinflation with diaphragmatic breathing, as well as providing some positive expiratory pressure can make fore a really useful set of exercises for someone who has impaired lung function, but make sure you get the order of operations correct!


Do you have any funny verbal cues you like to use for diaphragm retraining? Tell me about them in the comments!

Blow Out the Candles…

If there is any treatment that I feel like gets used in a cookie-cutter fashion, it’s pursed-lip breathing. As much as I hate seeing this technique used for every single patient that has shortness of breath, it does have clinical usefulness. So let’s talk about how to implement pursed-lip breathing properly based on patient presentationContinue reading “Blow Out the Candles…”

According to the Scientific Community, It’s Time for Change

If you couldn’t tell, I’ve been hinting at this for a while. In several posts over the past few months, there has been discussion amongst the medical community that has created controversy regarding the mode of transmission of COVID-19. Many providers of all disciplines have been very concerned about their contraction rates and that ofContinue reading “According to the Scientific Community, It’s Time for Change”

ABGs (Part 1)

This is the first post in a two-part series about understanding and interpreting arterial blood gases! If you want the rest of the post, you’ll have to check back next week! I’m getting real science-y again! So, just a heads up: my undergraduate degree was in chemistry. Kind of by accident, I took a lotContinue reading “ABGs (Part 1)”

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References:

Button, B. M., Holland, A. E., Sherburn, M. S., Chase, J., Wilson, J. W., & Burge, A. T. (2019). Prevalence, impact and specialised treatment of urinary incontinence in women with chronic lung disease. Physiotherapy105(1), 114–119. https://doi.org/10.1016/j.physio.2018.07.006

Lee, D. (2019). Butt-grippers, Back-grippers and Chest Grippers. Retrieved from https://dianeleephysio.com/education/butt-grippers-back-grippers-and-chest-grippers/

Follow @DoctorBthePT on Twitter for regular updates!

Postural Drainage

We’ve all seen that dreaded picture in our textbooks… All the human figures laying in so many different positions with pillows and tables tilted all over… and I very clearly remember thinking, “How on earth am I supposed to remember all of those?” Well, good news. You really don’t have to. It’s great if you do, don’t get me wrong, but for the purpose of general patient care, you can take a less specific approach and still have good results. Many of those positions cannot be achieved without a hospital bed unless you have a young, strong, and flexible patient, and if your patients is that young, strong, and flexible, they probably don’t need postural drainage.

Chest Physiotherapy (CPT) and Postural Drainage Positions | Respiratory  therapy student, Physical therapy assistant, Physiotherapy
Source

Yea, let’s be honest. Some of those are just not going to happen in any setting other than a hospital. So, if you don’t work in a hospital, how can you hope to achieve postural drainage? Good news, it’s no where near as difficult as it may seem. There is one solid rule to follow: Put the bad sound on top.


Step 1: Auscultate

As with so many cardiovascular and pulmonary interventions I’ve discussed, you have to know what you are listening for before you can ever hope to position someone properly. You can read all about lung auscultation in this post to get the details on that. But, basically, you have to auscultate to identify a segment that requires drainage.

Step 2: Identify the Segment

If you auscultated properly, you can compare the points you auscultated to an image for the underlying lung anatomy and figure out which segment is giving you adventitious sounds. If you you can’t google the picture really quick on your phone or computer, at least identify the lung side (right or left) and general lobe (superior, middle, lower). Remember, the left lung doesn’t have a middle lobe. Please don’t ever say you are assessing or treating the left middle lobe. It doesn’t exist.

Step 3: Put the Bad Sound on Top

Position the patient in a comfortable way so that the portion of the lung that produced the adventitious sound on auscultation is at the highest point of the body. Many of these can be achieved laying down in some way, but it will usually mean positioning the patient in sidelying at the very least. As you can see above, 6 of the 12 positions have some component of sidelying. So, if you are getting your patient in sidelying with the lung making bad sounds on top, you’ll make something better. Use as many pillows as it takes.


That’s it! Not so bad, right??? But you can’t stop there. Postural drainage positions are great and all, but, as with most other pulmonary interventions, they should not be used in isolation. While you have your patient in the drainage position, apply some other interventions to improve secretion mobility and clearance. Postural drainage positions should continue after your chosen interventions to facilitate further secretion clearance, about 10 minutes or so after you are done. Here is a list of ideas:

And, if you think about it, some of those positions probably won’t be needed by the majority of our patients. Sure, there are people with hypersecretion diseases, like cystic fibrosis, that may require drainage from all segments of their lungs on a regular basis. But your typical patient with COPD may only need a segment or two at a time, and may only be able to tolerate a segment or two at a time. And you can always refer back to your favorite diagram if you need specifics!

If we put it in the scope of the bigger picture, most of our patients are upright and sitting or standing during the day. Some are supine. If your patient is spending the majority of the day in standing or sitting already, they will likely not require that position for drainage, as the apical and anterior segments being drained all day long. If they are supine all day, they won’t likely need the anterior segments drained because they have been draining all day. You’ll be much more likely to need to opposite postures to address the bases, as secretions get trapped in them more easily due to their dependent position. Smaller segments such as the lingular segment may need more attention than others due to the position and somewhat closed off nature of the anatomy.


You’ll also need to remember that, although these may seem like simple positional changes, they are not for everyone. There is a reason this is part of skilled rehab intervention. Positioning is far more important than many give it credit for. Here’s some examples:

ICP Concerns – Patients who have concerns regarding intracranial pressure monitoring, such as those with recent neurosurgery, brain injury, or placement of an ICP bolt for any reason should not be inverted for postural drainage.

Shortness Of Breath (Dyspnea)
Source

Orthopnea – patients with cardiac conditions in addition to pulmonary may have difficulty breathing when laying supine. This is typical for patients in advanced heart failure and some patients who have COPD. This will definitely interfere with your positioning, but you can compensate using pillows or power bed controls to give them a little elevation to their comfort.

Prone Positioning – patients with respiratory conditions may initially be very hesitant to lie in prone as they feel it will restrict their breathing, especially if pillows are used around their face or head. However, we know that prone positioning actually increases ventilation, so physiologically, this is not a concern. Patients may still not tolerate this positioning due to fear or anxiety. Note that patients who are proned (such as when inpatient ICU status while ventilated) are typically sedated because this is a taxing posture for people who have difficulty breathing. Don’t expect a high tolerance in the begining.


All in all, I’ve had even the most severe end-stage COPD patients in prone over the edge of their beds with their hands on the floor to facilitate secretion mobilization while on supplemental oxygen with their spouse performing vibration. It can be done. And you can do it, too!


I feel like that superior lingular segment is the culprit 75% of the time. Do you find yourself regularly treating a specific lung segment? Tell me which one in the comments!

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More form the Pulmonary Rehab Toolbox…

Aerosol Generating Procedures

The long awaited clarification on aerosol generating procedures for physical therapists and physical therapist assistants has finally dropped! The APTA just released its professional guidelines for what portions of physical therapist and physical therapist assistant care equates to an aerosol generating procedures, therefore requiring increased PPE for procedure performance to ensure clinician safety. On AprilContinue reading “Aerosol Generating Procedures”

The Duet Device

Have you seen an Acapella Duet before? This is another one of those things I wish I could hand out to maybe half of my patients. A Duet device is a special kind of Positive Expiratory Pressure (PEP) that provides resistive oscillatory pressure to exhalation which promotes increased lung volumes due to re-inflation of collapsedContinue reading “The Duet Device”

FEV1

I spent some really great times as an educator in a heart and lung transplant program at a large hospital system. Specifically, I was a therapy educator. I taught PTs, PTAs, OTs, COTAs, and SLPs what they needed to know to safely provide rehab to patient after heart and lung transplants. I saw so manyContinue reading “FEV1”

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References:

Fink J. B. (2002). Positioning versus postural drainage. Respiratory care47(7), 769–777.

Hewitt, N., Bucknall, T., & Faraone, N. M. (2016). Lateral positioning for critically ill adult patients. The Cochrane database of systematic reviews2016(5), CD007205. https://doi.org/10.1002/14651858.CD007205.pub2

Follow @DoctorBthePT on Twitter for regular updates!

I’ll Huff and I’ll Puff… But the Huff Will Be More Effective

What is a huff? Huffing is one of the more difficulty techniques to learn for airway clearance. I find that most of my patients have trouble mustering the strength to perform a good solid huff. It’s not just that it is unfamiliar, but also that it takes diaphragm strength that they just don’t have. But when they nail it, it is so effective! So I keep teaching it even though it’s hard.

Huffing is part of the Forced Expiratory Techniques (FET). The name pretty much describes itself. You force an exhalation to mobilize secretions. Like many other airway clearance techniques, they are better when used in conjunction with other techniques, not independently.

So, along that line, when used as an FET, huff tend to be followed by diaphragmatic breathing or deep breathing cycles, or huffs may be incorporated in to the active cycle of breathing to achieve a similar outcome. There are several variations of the ACBT that can be implemented, but you can read about the standard one here. FET is also a great technique to place in between bouts of vibration or percussion, as evidence shows that this combination is significantly more effective when performed together, especially when used for long term management.

I’ve found the best way to teach it is to just show them. It’s kind of a silly sound and people tend to be a bit shy about performing it alone. I show them first so I’m the one to look silly, and then we do it together. When I teach a patient to huff as part of their FET home program, there are a few cues I like to use:

  1. The Ho, Ho, Ho
    • I ask the patient, “What does Santa Claus say?” Now I will admit that some people (it’s rare) do actually get this wrong and say “Merry Christmas”. So, I’ll give them the answer. Then they say, “Ho, Ho, Ho”.
    • Then I ask them to say it again but pretend Santa has smoked for 40 years.
    • This pretty much always works and is easy for a patient or caregiver to remember.
  2. The Culturally Sensitive Approach
    • I ask the patient to force a wheeze using their belly muscles to push air forcefully.
    • As long as I’m also demonstrating at the same time, this also almost always works. It does tend to require cues for increased force production.
Huff

Here’s what a decent huff sounds like:


What’s the difference between a huff and a cough?

That’s a really simple answer (not really my style, right?). The difference between a huff and a cough is an open glottis. Holding the glottis open lets air flow freely through the trachea, producing the airy huff sound.


Why not just cough?

Well, coughing is exhausting. My patients cough all day as it is and they really don’t want to cough more than they have to. They typically already have irritated airways from coughing or supplemental oxygen. Of course I also teach them energy conservation when it comes to coughing, but sometimes we need something a little less aggressive that can be performed more frequently for airway clearance. Huffing is equally effective at clearing secretions when compared to coughing, but is less strenuous when performed properly.

In some patients, too much huffing can result in collapse of lung tissue due to the back pressure against the glottis. Huffing doesn’t generate such high pressure, so the risk of collapse is less.


Making it personal…

Another variation you can play with is huffing at different lung volumes. Huffing at high lung volumes (when your lungs are mostly full of air already) helps to mobilize secretions in the peripheral regions of your lungs because the air has filled up those spaces and you utilize the huff the vibrate that air. Huffing at low lung volumes (when there isn’t much air in your lungs) helps to focus secretion mobilization to the larger more central airways to move those gathered secretions out.

You would typically want to huff at higher volumes first to mobilize peripheral secretions inward and then huff at lower volumes to mobilize what you’ve collected so it can be expelled. However, that depends on what the patient needs. If you are hearing ronchi on your auscultation, you’d probably want to huff at low volumes to clear the larger central airways before anything else.

Take a listen to the different between the two. When huffing at high lung volumes, you have your patient take a deep breath in and then huff it out. But, when huffing at low lung volumes, you have the patient breathe in and then most of the way out before the huff.

Huff at High Lung Volume
Huff at Low Lung Volume

Remember, FETs are aerosol-generating procedures, so be sure you and your patient are masked and in proper PPE. Like I said, these techniques are effective, so you’ll probably need some tissues handy also, or a cup if you are needing to collect a sample. And, as always, don’t forget to auscultate afterwards to assess progress!


Very useful and very effective, the best kind of tool to have in your toolbox! What is your favorite Pulmonary Rehab Tool we have discussed so far? Tell me in the comments!

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References:

D’Abrosca F, Garabelli B, Savio G. (2017). Comparing airways clearance techniques in chronic obstructive pulmonary disease and bronchiectasis: positive expiratory pressure or temporary positive expiratory pressure? A retrospective study. Brazilian Journal of Physical Therapy. 21(1):15-23. DOI: 10.1016/j.bjpt.2016.12.001.

Fink J. B. (2007). Forced expiratory technique, directed cough, and autogenic drainage. Respiratory care52(9), 1210–1223. Retrieved from http://rc.rcjournal.com/content/52/9/1210/tab-pdf

McIlwaine, M., Bradley, J., Elborn, J. S., & Moran, F. (2017). Personalising airway clearance in chronic lung disease. European respiratory review : an official journal of the European Respiratory Society26(143), 160086. https://doi.org/10.1183/16000617.0086-2016

Torres-Sánchez, I., Cruz-Ramírez, R., Cabrera-Martos, I., Díaz-Pelegrina, A., & Valenza, M. C. (2017). Results of Physiotherapy Treatments in Exacerbations of Chronic Obstructive Pulmonary Disease: A Systematic Review. Physiotherapy Canada. Physiotherapie Canada69(2), 122–132. https://doi.org/10.3138/ptc.2015-78

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